Science Daily 的報導指出用反轉病毒解釋人類進化樹碰到了巨大麻煩。
1。首先那些RNA片段的插入點在各猿類中遠不是同一個位置。
2。第二這些插入的RNA--DNA片段並非垃圾基因，而是新物種的重要基因組成部分 (當然原文從進化論角度愣解釋成是為進化論界被受爭議的史蒂芬古得的間斷平衡理論提供依據：即沒有過渡物種，只有突變後形成的全新物種，而這些病毒RNA的插入似乎起了不可替代的作用)
3。病毒譜系分析發現人類進化樹遠不是JTY所說的那麼清晰。有很多“竄種”的現象


因此，用反轉病毒解釋人類從猿到人的進化分支麻煩多多。

實驗觀測和預先設想的理論模型(進化樹)竟然100%全部吻合，分毫不差。呵呵，常識告訴我這世界上恐怕沒那麼便宜的事。一般這種too good to be true 的情況基本上不是更改、故意遺漏觀測結果，就是或者將實驗數無限誇大，愣往預先設想好的理論湊。

以下是原文摘錄。

(ZT)

In a new study, Evan Eichler and colleagues scanned finished chimpanzee genome sequence for endogenous retroviral elements, and found one (called PTERV1) that does not occur in humans. Searching the genomes of a subset of apes and monkeys revealed that the retrovirus had integrated into the germline of African great apes and Old World monkeys—but did not infect humans and Asian apes (orangutan, siamang, and gibbon). This undermines the notion that an ancient infection invaded an ancestral primate lineage, since great apes (including humans) share a common ancestor with Old World monkeys.

Eichler and colleagues found over 100 copies of PTERV1 in each African ape (chimp and gorilla) and Old World monkey (baboon and macaque) species. The authors compared the sites of viral integration in each of these primates and found that few if any of these insertion sites were shared among the primates. It appears therefore that the sequences have not been conserved from a common ancestor, but are specific to each lineage.