Science Daily 的报导指出用反转病毒解释人类进化树碰到了巨大麻烦。
1。首先那些RNA片段的插入点在各猿类中远不是同一个位置。
2。第二这些插入的RNA--DNA片段并非垃圾基因，而是新物种的重要基因组成部分 (当然原文从进化论角度愣解释成是为进化论界被受争议的史蒂芬古得的间断平衡理论提供依据：即没有过渡物种，只有突变后形成的全新物种，而这些病毒RNA的插入似乎起了不可替代的作用)
3。病毒谱系分析发现人类进化树远不是JTY所说的那么清晰。有很多“窜种”的现象


因此，用反转病毒解释人类从猿到人的进化分支麻烦多多。

实验观测和预先设想的理论模型(进化树)竟然100%全部吻合，分毫不差。呵呵，常识告诉我这世界上恐怕没那么便宜的事。一般这种too good to be true 的情况基本上不是更改、故意遗漏观测结果，就是或者将实验数无限夸大，愣往预先设想好的理论凑。

以下是原文摘录。

(ZT)

In a new study, Evan Eichler and colleagues scanned finished chimpanzee genome sequence for endogenous retroviral elements, and found one (called PTERV1) that does not occur in humans. Searching the genomes of a subset of apes and monkeys revealed that the retrovirus had integrated into the germline of African great apes and Old World monkeys—but did not infect humans and Asian apes (orangutan, siamang, and gibbon). This undermines the notion that an ancient infection invaded an ancestral primate lineage, since great apes (including humans) share a common ancestor with Old World monkeys.

Eichler and colleagues found over 100 copies of PTERV1 in each African ape (chimp and gorilla) and Old World monkey (baboon and macaque) species. The authors compared the sites of viral integration in each of these primates and found that few if any of these insertion sites were shared among the primates. It appears therefore that the sequences have not been conserved from a common ancestor, but are specific to each lineage.